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This IGF-1 independence is the single most important experimental advantage of the AOD 9604 10mg research compound for researchers designing metabolic studies: the absence of IGF-1 elevation means that downstream anabolic, mitogenic, or glucose-modulating confounds associated with full-length hGH are not present in AOD-9604 experimental models

doi: 10.1016/j.bbi.2010.09.002

A novel LIPE nonsense mutation found using exome sequencing in siblings with late-onset familial partial lipodystrophy

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Pathogenic SYNGAP1 mutations impair cognitive development by disrupting maturation of dendritic spine synapses